New Paper: Role of IRF8 in immune cells functions, protection against infections, and susceptibility to inflammatory diseases.
Study finds that artificially sweetened beverages consumption during pregnancy impacts mother’s microbiota and might lead to higher infant BMI.
New Paper: Consumption of Artificially Sweetened Beverages During Pregnancy Impacts Infant Gut Microbiota and Body Mass Index
Study finds that artificially sweetened beverages consumption during pregnancy impacts mother’s microbiota and might lead to higher infant BMI.
New Paper: Effect of Chicory Inulin-Type Fructan-Containing Snack Bars on the Human Gut Microbiota in Low Dietary Fiber Consumers in a Randomized Crossover Trial
Effect of chicory inulin-type fructan–containing snack bars on the human gut microbiota in low dietary fiber consumers in a randomized crossover trial.
New Paper: Clostridioides difficile Colonization Is Differentially Associated With Gut Microbiome Profiles by Infant Feeding Modality at 3–4 Months of Age
Congratulations to our Platform 2 Lead Dr. Anita Kozyrskyj on this recent publication! Abstract Colonization with Clostridioides difficile occurs in up to half of infants under the age of 3 months, is strongly influenced by feeding modality and is largely asymptomatic. In spite of this, C. difficile‘s presence has been associated with susceptibility to chronic disease later in […]
New Book: Microbiota in Health and Disease: From Pregnancy to Childhood
In 2017, Editors Pamela D. Browne, Eric Claassen and Michael D. Cabana published a book about the Microbiota in health and disease: from pregnancy to childhood in Wageningen Academic Publishers. Microbiota in health and disease: from pregnancy to childhood aims to provide a state-of-the-art overview in the field of infant and child microbiota and their role in health, […]
New Paper: Microbiota-Derived Peptide Mimics Drive Lethal Inflammatory Cardiomyopathy
Congratulations to IMPACTT Director and Platform 1 Lead Dr. Kathy McCoy, as well as Education & Mentorship Lead Dr. Markus Geuking, on this recent publication! Abstract Myocarditis can develop into inflammatory cardiomyopathy through chronic stimulation of myosin heavy chain 6–specific T helper (TH)1 and TH17 cells. However, mechanisms governing the cardiotoxicity programming of heart-specific T […]
